Overview
- 1 May 2003
- journal article
- review article
- Published by Wolters Kluwer Health in Journal of the American Society of Nephrology
- Vol. 14 (5) , 1355-1357
- https://doi.org/10.1097/01.asn.0000065608.37756.58
Abstract
No abstract availableThis publication has 10 references indexed in Scilit:
- Protein Kinase C β Inhibition Attenuates the Progression of Experimental Diabetic Nephropathy in the Presence of Continued HypertensionDiabetes, 2003
- Reversibility of established diabetic glomerulopathy by anti-TGF-β antibodies in db/db miceBiochemical and Biophysical Research Communications, 2003
- Cellular Basis of Diabetic NephropathyDiabetes, 2002
- Inhibiting albumin glycation in vivo ameliorates glomerular overexpression of TGF-β1Kidney International, 2002
- Biochemistry and molecular cell biology of diabetic complicationsNature, 2001
- Hyperglycemia-induced mitochondrial superoxide overproduction activates the hexosamine pathway and induces plasminogen activator inhibitor-1 expression by increasing Sp1 glycosylationProceedings of the National Academy of Sciences, 2000
- Long-term prevention of renal insufficiency, excess matrix gene expression, and glomerular mesangial matrix expansion by treatment with monoclonal antitransforming growth factor-β antibody indb/dbdiabetic miceProceedings of the National Academy of Sciences, 2000
- GLUT1 and TGF-β: the link between hyperglycaemia and diabetic nephropathyNephrology Dialysis Transplantation, 1999
- Increased Renal Production of Transforming Growth Factor-β1 in Patients with Type II DiabetesDiabetes, 1997
- Cell and Matrix Components of the Glomerular Mesangium in Type I DiabetesDiabetes, 1992