AN EXPERIMENTALLY PRODUCED PREMATURE SYSTOLIC ARHYTHMIA (PULSUS BIGEMINUS) IN RABBITS

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Abstract
Weak faradization of the motor area of the cerebrum, superior colliculi, olfactory bulbs, hippocampus and habenula bodies (2 animals only) did not produce premature systoles. Osmotic-chemical excitations of the deeper portions of the olfactory bulbs, motor area and hippocampus seldom resulted in premature systoles. The few instances in which a premature systolic arhythmia followed strong faradization of the superior colliculus or some of the osmotic-chemical stimulations, can in all probability be attributed to the accompanying enormous rise in blood pressure. A premature systolic arhythmia was elicited in 3 rabbits from faradization of the hypothalamus. The premature systolic arhythmias obtained from benzol insufflation after the mesencephalon had been transected, demonstrates a trigeminal connection to an efferent center below the diencephalon for inducing this arhythmia. Several observations not included in this problem, but showing the effects of faradic and osmotic-chemical stimulation of various effective areas in the brain are listed.

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