Targeted Proteomic Analysis of 14-3-3ς, a p53 Effector Commonly Silenced in Cancer
Open Access
- 1 June 2005
- journal article
- Published by Elsevier in Molecular & Cellular Proteomics
- Vol. 4 (6) , 785-795
- https://doi.org/10.1074/mcp.m500021-mcp200
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Downregulation of 14-3-3σ in ovary, prostate and endometrial carcinomas is associated with CpG island methylationLaboratory Investigation, 2005
- Prostate cancer is characterized by epigenetic silencing of 14-3-3σ expressionOncogene, 2004
- The 14-3-3 cancer connectionNature Reviews Cancer, 2003
- Analysis of 14-3-3σ expression in hyperproliferative skin diseases reveals selective loss associated with CpG-methylation in basal cell carcinomaOncogene, 2003
- Frequent and histological type-specific inactivation of 14-3-3σ in human lung cancersOncogene, 2002
- The G2/M Regulator 14-3-3ς Prevents Apoptosis through Sequestration of BaxJournal of Biological Chemistry, 2001
- Frequent hypermethylation of CpG islands and loss of expression of the 14-3-3 σ gene in human hepatocellular carcinomaOncogene, 2000
- High frequency of hypermethylation at the 14-3-3 σ locus leads to gene silencing in breast cancerProceedings of the National Academy of Sciences, 2000
- 14-3-3σ is required to prevent mitotic catastrophe after DNA damageNature, 1999
- Is a p53-Regulated Inhibitor of G2/M ProgressionMolecular Cell, 1997