Partial resistance to anticoagulation after streptokinase treatment for acute myocardial infarction
- 1 July 1990
- journal article
- research article
- Published by Elsevier in The American Journal of Cardiology
- Vol. 66 (1) , 28-30
- https://doi.org/10.1016/0002-9149(90)90730-o
Abstract
No abstract availableThis publication has 14 references indexed in Scilit:
- Intravenous streptokinase for acute myocardial infarction. Effects on global and regional systolic function.Circulation, 1988
- Streptokinase-induced, antibody-mediated platelet aggregation: A potential cause of clot propagation in vivoJournal of the American College of Cardiology, 1988
- Thrombolytic Therapy: Current StatusNew England Journal of Medicine, 1988
- Thrombolytic Therapy: Current StatusNew England Journal of Medicine, 1988
- Marked platelet activation in vivo after intravenous streptokinase in patients with acute myocardial infarction.Circulation, 1988
- Paradoxic elevation of fibrinopeptide A after streptokinase: Evidence for continued thrombosis despite intense fibrinolysisJournal of the American College of Cardiology, 1987
- Role of heparin after intravenous thrombolytic therapy for acute myocardial infarctionThe American Journal of Cardiology, 1987
- Importance of continued activation of thrombin reflected by fibrinopeptide A to the efficacy of thrombolysisJournal of the American College of Cardiology, 1986
- Prevention of Myocardial Damage in Acute Myocardial Ischemia by Early Treatment with Intravenous StreptokinaseNew England Journal of Medicine, 1985
- Nonsurgical reperfusion in evolving myocardial infarctionJournal of the American College of Cardiology, 1983