ATM-dependent Phosphorylation of Human Rad9 Is Required for Ionizing Radiation-induced Checkpoint Activation
Open Access
- 1 May 2001
- journal article
- Published by Elsevier
- Vol. 276 (19) , 16580-16586
- https://doi.org/10.1074/jbc.m008871200
Abstract
No abstract availableKeywords
This publication has 66 references indexed in Scilit:
- Retention of the Human Rad9 Checkpoint Complex in Extraction-resistant Nuclear Complexes after DNA DamagePublished by Elsevier ,2000
- Physical Interactions among Human Checkpoint Control Proteins HUS1p, RAD1p, and RAD9p, and Implications for the Regulation of Cell Cycle ProgressionGenomics, 2000
- Human DNA Damage Checkpoint Protein hRAD9 Is a 3′ to 5′ ExonucleasePublished by Elsevier ,2000
- hRAD17, a structural homolog of the Schizosaccharomyces pombe RAD17 cell cycle checkpoint gene, stimulates p53 accumulationOncogene, 1999
- RAD1,a Human Structural Homolog of theSchizosaccharomyces pombe RAD1Cell Cycle Checkpoint GeneGenomics, 1998
- A Human and Mouse Homolog of theSchizosaccharomyces pombe rad1+Cell Cycle Checkpoint Control GeneGenomics, 1998
- DNA Damage and Checkpoint PathwaysCell, 1998
- Duplication of ATR inhibits MyoD, induces aneuploidy and eliminates radiation-induced G1 arrestNature Genetics, 1998
- THE GENETIC DEFECT IN ATAXIA-TELANGIECTASIAAnnual Review of Immunology, 1997
- Phosphorylation of the retinoblastoma gene product is modulated during the cell cycle and cellular differentiationCell, 1989