JAK2/STAT3, Not ERK1/2, Mediates Interleukin-6-induced Activation of Inducible Nitric-oxide Synthase and Decrease in Contractility of Adult Ventricular Myocytes

This publication has 43 references indexed in Scilit:

• Interleukin-6-induced Tethering of STAT3 to the LAP/C/EBPβ Promoter Suggests a New Mechanism of Transcriptional Regulation by STAT3
  Journal of Biological Chemistry, 2001
• Myocardial Ischemia Activates the JAK–STAT Pathway Through Angiotensin II Signaling in in vivo Myocardium of Rats
  Journal of Molecular and Cellular Cardiology, 2001
• Roles of STAT3 in mediating the cell growth, differentiation and survival signals relayed through the IL-6 family of cytokine receptors
  Oncogene, 2000
• Cytokines and JAK–STAT Signaling
  Experimental Cell Research, 1999
• Factors That Control Sarcoplasmic Reticulum Calcium Release in Intact Ventricular Myocytes^a
  Annals of the New York Academy of Sciences, 1998
• Cytokine profile in chronic cardiac failure
  European Journal of Clinical Investigation, 1996
• Myocardium is a major source of proinflammatory cytokines in patients undergoing cardiopulmonary bypass
  The Journal of Thoracic and Cardiovascular Surgery, 1996
• Regulation of Cytokine-inducible Nitric Oxide Synthase in Cardiac Myocytes and Microvascular Endothelial Cells: ROLE OF EXTRACELLULAR SIGNAL-REGULATED KINASES 1 AND 2 (ERK1/ERK2) AND STAT1α
  Published by Elsevier ,1996
• Negative Inotropic Effects of Cytokines on the Heart Mediated by Nitric Oxide
  Science, 1992
• Serum interleukin 6 levels become elevated in acute myocardial infarction
  Journal of Molecular and Cellular Cardiology, 1992