The c-Jun transcription factor – bipotential mediator of neuronal death, survival and regeneration
- 1 May 1997
- journal article
- review article
- Published by Elsevier in Trends in Neurosciences
- Vol. 20 (5) , 227-231
- https://doi.org/10.1016/s0166-2236(96)01000-4
Abstract
No abstract availableKeywords
This publication has 66 references indexed in Scilit:
- c-JUN, KROX-24, and c-FOS Expression in Hippocampal Grafts Placed in Excitotoxic Hippocampal Lesions of the RatExperimental Neurology, 1995
- Comparison of c‐jun, junB, and junD mRNA expression and protein in the rat dorsal root ganglia following sciatic nerve transectionJournal of Neuroscience Research, 1995
- The Bcl-2 family of proteins, and the regulation of neuronal survivalTrends in Neurosciences, 1995
- Gap‐43 immunoreactivity and axon regeneration in retinal ganglion cells of the ratJournal of Neurobiology, 1994
- Oncogenes and cell deathCurrent Opinion in Genetics & Development, 1994
- Injury‐associated induction of GAP‐43 expression displays axon branch specificity in rat dorsal root ganglion neuronsJournal of Neurobiology, 1993
- Long-term increase in the levels of c-jun mRNA and jun protein-like immunoreactivity in motor and sensory neurons following axon damageNeuroscience Letters, 1991
- Expression of the growth-associated protein GAP-43 in adult rat retinal ganglion cells following axon injuryNeuron, 1991
- Regeneration of long spinal axons in the ratJournal of Neurocytology, 1984
- Regeneration and retrograde degeneration of axons in the rat optic nerveJournal of Neurocytology, 1982