Inflammatory conditions induce gap junctional communication between rat Kupffer cells both in vivo and in vitro
- 30 June 2007
- journal article
- Published by Elsevier in Cellular Immunology
- Vol. 247 (2) , 103-110
- https://doi.org/10.1016/j.cellimm.2007.08.001
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- HIV-tat induces formation of an LRP–PSD-95– NMDAR–nNOS complex that promotes apoptosis in neurons and astrocytesProceedings of the National Academy of Sciences, 2007
- Injury of skeletal muscle and specific cytokines induce the expression of gap junction channels in mouse dendritic cellsJournal of Cellular Physiology, 2007
- Pannexin-1 mediates large pore formation and interleukin-1β release by the ATP-gated P2X7 receptorThe EMBO Journal, 2006
- CCL2/Monocyte Chemoattractant Protein-1 Mediates Enhanced Transmigration of Human Immunodeficiency Virus (HIV)-Infected Leukocytes across the Blood–Brain Barrier: A Potential Mechanism of HIV–CNS Invasion and NeuroAIDSJournal of Neuroscience, 2006
- Shedding of PECAM-1 during HIV infection: a potential role for soluble PECAM-1 in the pathogenesis of NeuroAIDSJournal of Leukocyte Biology, 2005
- Endotoxin unmasks the role of gap junctions in the liverBiochemical and Biophysical Research Communications, 2004
- Kupffer cell inactivation prevents lipopolysaccharide-induced structural changes in the rat liver sinusoid: An electron-microscopic studyHepatology, 1996
- Posttranscriptional regulation of connexin 32 expression in liver during acute inflammationJournal of Cellular Physiology, 1996
- Structure and Function of Sinusoidal Lining Cells in the LiverToxicologic Pathology, 1996
- Reversible inhibition of intercellular junctional communication by glycyrrhetinic acidBiochemical and Biophysical Research Communications, 1986