Abstract
After the initial attack of herpes labialis, herpes simplex virus type 1 is thought to remain latent in the trigeminal ganglion and to be reactivated by various types of stress.2 Although things such as fever, upper respiratory tract infections, ultraviolet light, and psychological stress are thought to trigger recurrence, the condition is variable and unpredictable in most patients, as is the extent and duration of each attack.3 Recurrent attacks are usually shorter and less painful than the original attack.