HMG-CoA reductase inhibition by atorvastatin reduces neointimal inflammation in a rabbit model of atherosclerosis
- 1 December 1998
- journal article
- Published by Elsevier in Journal of the American College of Cardiology
- Vol. 32 (7) , 2057-2064
- https://doi.org/10.1016/s0735-1097(98)00487-2
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- Tumor Necrosis Factor-α Activates Smooth Muscle Cell Migration in Culture and Is Expressed in the Balloon-Injured Rat AortaArteriosclerosis, Thrombosis, and Vascular Biology, 1997
- Increased expression of matrix metalloproteinases and matrix degrading activity in vulnerable regions of human atherosclerotic plaques.Journal of Clinical Investigation, 1994
- Genetic evidence for a common pathway mediating oxidative stress, inflammatory gene induction, and aortic fatty streak formation in mice.Journal of Clinical Investigation, 1994
- Characterization of the relative thrombogenicity of atherosclerotic plaque components: Implications for consequences of plaque ruptureJournal of the American College of Cardiology, 1994
- Genetic control of inflammatory gene induction and NF-kappa B-like transcription factor activation in response to an atherogenic diet in mice.Journal of Clinical Investigation, 1993
- HMG CoA reductase inhibitors. In vivo effects on carotid intimal thickening in normocholesterolemic rabbits.Arteriosclerosis and Thrombosis: A Journal of Vascular Biology, 1993
- Effects on coronary artery disease of lipid-lowering diet, or diet plus cholestyramine, in the St Thomas' Atherosclerosis Regression Study (STARS)The Lancet, 1992
- The Pathogenesis of Coronary Artery Disease and the Acute Coronary SyndromesNew England Journal of Medicine, 1992
- Monocyte chemoattractant protein-1 in human atheromatous plaques.Journal of Clinical Investigation, 1991
- New Rabbit Atherosclerosis Model for the Investigation of Transluminal AngioplastyInvestigative Radiology, 1982