Constitutive expression of arginase in microvascular endothelial cells counteracts nitric oxide‐mediated vasodilatory function
- 12 March 2001
- journal article
- Published by Wiley in The FASEB Journal
- Vol. 15 (7) , 1264-1266
- https://doi.org/10.1096/fj.00-0681fje
Abstract
No abstract availableFunding Information
- National Heart, Lung, and Blood Institute (HL‐55524, HL‐48179, K02HL03693)
This publication has 30 references indexed in Scilit:
- The Involvement of Tyrosine Kinases, Cyclic AMP/Protein Kinase A, and p38 Mitogen-Activated Protein Kinase in IL-13-Mediated Arginase I Induction in Macrophages: Its Implications in IL-13-Inhibited Nitric Oxide ProductionThe Journal of Immunology, 2000
- IL-4 and IL-13 upregulate arginase I expression by cAMP and JAK/STAT6 pathways in vascular smooth muscle cellsAmerican Journal of Physiology-Cell Physiology, 2000
- Arginase modulates nitric oxide production in activated macrophagesAmerican Journal of Physiology-Heart and Circulatory Physiology, 1998
- Human type II arginase: sequence analysis and tissue-specific expressionGene, 1997
- Comparative properties of arginasesComparative Biochemistry and Physiology Part B: Biochemistry and Molecular Biology, 1996
- Acidosis-Induced Coronary Arteriolar Dilation Is Mediated by ATP-Sensitive Potassium Channels in Vascular Smooth MuscleCirculation Research, 1996
- Nitric oxide synthase isozymes. Characterization, purification, molecular cloning, and functions.Hypertension, 1994
- L-arginine is the physiological precursor for the formation of nitric oxide in endothelium-dependent relaxationBiochemical and Biophysical Research Communications, 1988
- Endothelium-derived relaxing factor produced and released from artery and vein is nitric oxide.Proceedings of the National Academy of Sciences, 1987
- Molecular cloning and nucleotide sequence of cDNA for human liver arginase.Proceedings of the National Academy of Sciences, 1987