Cytosolic Ca2+ buffering, a cell property that in some neurons markedly decreases during aging, has a protective effect against nmda/nitric oxide-induced excitotoxicity
- 3 July 1996
- journal article
- Published by Elsevier in Life Sciences
- Vol. 59 (5-6) , 389-397
- https://doi.org/10.1016/0024-3205(96)00317-7
Abstract
No abstract availableKeywords
This publication has 21 references indexed in Scilit:
- Cytosolic Ca2+ Binding Proteins during Rat Brain Ageing: Loss of Calbindin and Calretinin in the Hippocampus, with no Change in the CerebellumEuropean Journal of Neuroscience, 1994
- Cell-permeant Ca2+ chelators reduce early excitotoxic and ischemic neuronal injury in vitro and in vivoNeuron, 1993
- Effects of calcium chelators on intracellular calcium and excitotoxicityNeuroscience Letters, 1993
- Evidence that CHP100 neuroblastoma cell death induced by N-methyl-d-aspartate involves l-arginine-nitric oxide pathway activationNeuroscience Letters, 1992
- Stable transfection of calbindin-D28k into the GH3 cell line alters calcium currents and intracellular calcium homeostasisNeuron, 1992
- Calcium-binding proteins in the nervous systemTrends in Neurosciences, 1992
- Loss of Calbindin-28K Immunoreactivity in Hippocampal Slices from Aged Rats: a Role for Calcium?European Journal of Neuroscience, 1991
- Evidence for calcium-reducing and excitoprotective roles for the calcium-binding protein calbindin-1328k in cultured hippocampal neuronsNeuron, 1991
- The Role of Glutamate Neurotoxicity in Hypoxic-Ischemic Neuronal DeathAnnual Review of Neuroscience, 1990
- Calcium transport and buffering in neuronsTrends in Neurosciences, 1988