Toll signaling: RIPping off the TNF pathway.
- 1 May 2004
- journal article
- editorial
- Published by Springer Nature in Nature Immunology
- Vol. 5 (5) , 472-474
- https://doi.org/10.1038/ni0504-472
Abstract
No abstract availableKeywords
This publication has 12 references indexed in Scilit:
- RIP1 is an essential mediator of Toll-like receptor 3–induced NF-κB activationNature Immunology, 2004
- Kinase RIP3 Is Dispensable for Normal NF-κBs, Signaling by the B-Cell and T-Cell Receptors, Tumor Necrosis Factor Receptor 1, and Toll-Like Receptors 2 and 4Molecular and Cellular Biology, 2004
- IFN-regulatory factor 3-dependent gene expression is defective in Tbk1 -deficient mouse embryonic fibroblastsProceedings of the National Academy of Sciences, 2003
- Toll/IL-1 Receptor Domain-Containing Adaptor Inducing IFN-β (TRIF) Associates with TNF Receptor-Associated Factor 6 and TANK-Binding Kinase 1, and Activates Two Distinct Transcription Factors, NF-κB and IFN-Regulatory Factor-3, in the Toll-Like Receptor SignalingThe Journal of Immunology, 2003
- Role of Adaptor TRIF in the MyD88-Independent Toll-Like Receptor Signaling PathwayScience, 2003
- Identification of Lps2 as a key transducer of MyD88-independent TIR signallingNature, 2003
- IKKε and TBK1 are essential components of the IRF3 signaling pathwayNature Immunology, 2003
- TICAM-1, an adaptor molecule that participates in Toll-like receptor 3–mediated interferon-β inductionNature Immunology, 2003
- Cutting Edge: A Novel Toll/IL-1 Receptor Domain-Containing Adapter That Preferentially Activates the IFN-β Promoter in the Toll-Like Receptor SignalingThe Journal of Immunology, 2002
- The Death Domain Kinase RIP Mediates the TNF-Induced NF-κB SignalImmunity, 1998