Scleroderma and Smads: Dysfunctional Smad family dynamics culminating in fibrosis
- 11 July 2002
- journal article
- review article
- Published by Wiley in Arthritis & Rheumatism
- Vol. 46 (7) , 1703-1713
- https://doi.org/10.1002/art.10413
Abstract
No abstract availableThis publication has 85 references indexed in Scilit:
- TGF-β induces apoptosis through Smad-mediated expression of DAP-kinaseNature Cell Biology, 2001
- Transforming Growth Factor-β Repression of Matrix Metalloproteinase-1 in Dermal Fibroblasts Involves Smad3Journal of Biological Chemistry, 2001
- Differential Expression of Transforming Growth Factor-β Receptors I and II and Activation of Smad 3 in Keloid FibroblastsPlastic and Reconstructive Surgery, 2001
- Decreased susceptibility to Fas-induced apoptosis of systemic sclerosis dermal fibroblastsArthritis & Rheumatism, 2001
- Expression of Smads during in Vitro Transdifferentiation of Hepatic Stellate Cells to MyofibroblastsBiochemical and Biophysical Research Communications, 2001
- Interaction and Functional Cooperation of NF-κB with SmadsJournal of Biological Chemistry, 2000
- Modulation of Endogenous Smad Expression in Normal Skin Fibroblasts by Transforming Growth Factor-βExperimental Cell Research, 2000
- A mechanism of repression of TGFbeta / Smad signaling by oncogenic RasGenes & Development, 1999
- Animal Model of Sclerotic Skin. I: Local Injections of Bleomycin Induce Sclerotic Skin Mimicking SclerodermaJournal of Investigative Dermatology, 1999
- Sp1 Is Required for the Early Response of α2(I) Collagen to Transforming Growth Factor-β1Journal of Biological Chemistry, 1997