Effects of oxidative stress on the expression of antioxidative defense enzymes in spontaneously hypertensive rat hearts

This publication has 24 references indexed in Scilit:

• Changes in mRNA levels of the sarcoplasmic/endoplasmic-reticulum Ca2+-ATPase isoforms in the rat soleus muscle regenerating from notexin-induced necrosis
  Biochemical Journal, 1996
• Myocardial Fibrosis and Stiffness With Hypertrophy and Heart Failure in the Spontaneously Hypertensive Rat
  Circulation, 1995
• The spontaneously hypertensive rat as a model of the transition from compensated left ventricular hypertrophy to failure
  Journal of Molecular and Cellular Cardiology, 1995
• The sera of spontaneously hypertensive rats contain agonistic auto-antibodies against the ??1-radrenoceptor
  Journal Of Hypertension, 1995
• Hypothesis: Apoptosis May be a Mechanism for the Transition to Heart Failure with Chronic Pressure Overload
  Journal of Molecular and Cellular Cardiology, 1994
• Adenosine triphosphate-sensitive potassium channel blocking agent ameliorates, but the opening agent aggravates, ischemia/reperfusion-induced injury: Heart function studies in nonfibrillatingt isolated hearts
  Journal of the American College of Cardiology, 1994
• Reoxygenation-induced cell damage of isolated neonatal rat ventricular myocytes can be reduced by chain-breaking antioxidants
  Free Radical Biology & Medicine, 1994
• Molecular Structure of a Functional Rat Gene for Manganese-containing Superoxide Dismutase
  American Journal of Respiratory Cell and Molecular Biology, 1991
• Diminished cardiac hypertrophy and muscle performance in older compared with younger adult rats with chronic atrioventricular block.
  Circulation Research, 1988
• Reperfusion-induced arrhythmias and oxygen-derived free radicals. Studies with "anti-free radical" interventions and a free radical-generating system in the isolated perfused rat heart.
  Circulation Research, 1986