Mechanisms of Resistance to Macrolides and Lincosamides: Nature of the Resistance Elements and Their Clinical Implications
Top Cited Papers
Open Access
- 15 February 2002
- journal article
- review article
- Published by Oxford University Press (OUP) in Clinical Infectious Diseases
- Vol. 34 (4) , 482-492
- https://doi.org/10.1086/324626
Abstract
Resistance to macrolides and lincosamides is increasingly reported in clinical isolates of gram-positive bacteria. The multiplicity of mechanisms ofKeywords
This publication has 74 references indexed in Scilit:
- Serotype 19F Multiresistant Pneumococcal Clone Harboring Two Erythromycin Resistance Determinants [ erm (B) and mef (A)] in South AfricaAntimicrobial Agents and Chemotherapy, 2001
- Identification of an erm (A) Erythromycin Resistance Methylase Gene in Streptococcus pneumoniae Isolated in GreeceAntimicrobial Agents and Chemotherapy, 2001
- Macrolide Resistance Conferred by Base Substitutions in 23S rRNAAntimicrobial Agents and Chemotherapy, 2001
- Induction of Ribosome Methylation in MLS-ResistantStreptococcus pneumoniaeby Macrolides and KetolidesMicrobial Drug Resistance, 1999
- Molecular cloning and functional analysis of a novel macrolide‐resistance determinant, mefA, from Streptococcus pyogenesMolecular Microbiology, 1996
- Insights into erythromycin action from studies of its activity as inducer of resistanceAntimicrobial Agents and Chemotherapy, 1995
- Erythromycin resistance by ribosome modificationAntimicrobial Agents and Chemotherapy, 1995
- Inducible erythromycin resistance in staphlyococci is encoded by a member of the ATP‐binding transport super‐gene familyMolecular Microbiology, 1990
- Clindamycin therapy of staphylococcus aureus endocarditisThe American Journal of Medicine, 1976
- THE SENSITIVITY OF STAPHYLOCOCCI AND OTHER WOUND BACTERIA TO ERYTHROMYCIN, OLEANDOMYCIN, AND SPIRAMYCINJournal of Clinical Pathology, 1959