THE REGULATION OF RESPIRATION

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Abstract
Low alveolar 0 increased the carotid flow of blood. Recovery followed upon breathing room air. Low alveolar O produced an initial decrease in femoral arterial flow, succeeded by a flow greater than the original. Readministration of room air produced a further rapid increase in flow followed by a decrease to the first level. NaCN (intravenous) produced an increase in carotid flow and an initial decrease in femoral, followed by increase during recovery. High alveolar 0 produced a lessened flow in both carotid and femoral arteries. These changes are not dependent on changes in blood pressure. It is believed that changes of common carotid blood flow are indicative of changes in brain flow and that head flow in general varies as the brain flow. The possibility of a central and peripheral acid mechanism of volume flow control, modified by the paralytic action of reduced oxidation, is proposed. The results are not in disagreement with the view that the metabolism of the vasomotor center may play a part in the control of volume flow of blood.

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