Control of Autoimmune Diabetes
- 14 January 2000
- journal article
- Published by American Association for the Advancement of Science (AAAS) in Science
- Vol. 287 (5451) , 191
- https://doi.org/10.1126/science.287.5451.191a
Abstract
No abstract availableThis publication has 11 references indexed in Scilit:
- Control of Autoimmune Diabetes in NOD Mice by GAD Expression or Suppression in β CellsScience, 1999
- GABAA receptors mediate inhibition of T cell responsesJournal of Neuroimmunology, 1999
- Two isoforms of glutamate decarboxylase: why?Trends in Pharmacological Sciences, 1998
- B lymphocytes are essential for the initiation of T cell-mediated autoimmune diabetes: analysis of a new "speed congenic" stock of NOD.Ig mu null mice.The Journal of Experimental Medicine, 1996
- Diabetogenic T-Cell ClonesDiabetes, 1996
- Immunological and Metabolic Effects of Prophylactic Insulin Therapy in the NOD-scid/scid Adoptive Transfer Model of IDDMDiabetes, 1996
- Differential Expression of GAD65 and GAD67 in Human, Rat, and Mouse Pancreatic IsletsDiabetes, 1993
- Immune response to glutamic acid decarboxylase correlates with insulitis in non-obese diabetic miceNature, 1993
- Spontaneous loss of T-cell tolerance to glutamic acid decarboxylase in murine insulin-dependent diabetesNature, 1993
- Localization of GAD-like immunoreactivity in the pancreas and stomach of the rat and mouseHistochemistry and Cell Biology, 1991