K+ currents of human T-lymphocytes are unaffected by Alzheimer's disease and amyloid β protein
- 1 January 1996
- journal article
- Published by Elsevier in Neuroscience Letters
- Vol. 202 (3) , 177-180
- https://doi.org/10.1016/0304-3940(95)12243-5
Abstract
No abstract availableKeywords
This publication has 12 references indexed in Scilit:
- Soluble β-Amyloid Induction of Alzheimer's Phenotype for Human Fibroblast K + ChannelsScience, 1994
- Similar age-related changes of free intracellular calcium in lymphocytes and central neurons: Effects of Alzheimer's diseaseArchiv Fur Psychiatrie Und Nervenkrankheiten, 1994
- Calcium-activated potassium channels in resting and activated human T lymphocytes. Expression levels, calcium dependence, ion selectivity, and pharmacology.The Journal of general physiology, 1993
- β‐Amyloid protein enhances the mitogen‐induced calcium response in circulating human lymphocytesFEBS Letters, 1993
- Intracellular calcium response is reduced in CD4+ lymphocytes in Alzheimer's disease and in older persons with down's syndromeNeurobiology of Aging, 1993
- Amyloid β peptides act directly on single neuronsNeuroscience Letters, 1993
- Detection of distinct isoform patterns of the β-amyloid precursor protein in human platelets and lymphocytesNeurobiology of Aging, 1992
- beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicityJournal of Neuroscience, 1992
- Ion Channels and Signal Transduction in LymphocytesAnnual Review of Physiology, 1990
- Prolonged Ca 2+ -Dependent Afterhyperpolarizations in Hippocampal Neurons of Aged RatsScience, 1984