The role of Bax in glutamate‐induced nerve cell death
Open Access
- 1 January 2001
- journal article
- research article
- Published by Wiley in Journal of Neurochemistry
- Vol. 76 (1) , 295-301
- https://doi.org/10.1046/j.1471-4159.2001.00035.x
Abstract
The role of the Bax gene product was examined in three forms of cortical nerve cell death in primary cultures. These include spontaneous cell death, oxidative glutamate toxicity, in which exogenous g...Keywords
This publication has 38 references indexed in Scilit:
- Lounging in a lysosome: the intracellular lifestyle of Coxiella burnetiiCellular Microbiology, 2007
- BCL-2 FAMILY: Regulators of Cell DeathAnnual Review of Immunology, 1998
- Mechanism of Cellular 3‐(4,5‐Dimethylthiazol‐2‐yl)‐2,5‐Diphenyltetrazolium Bromide (MTT) ReductionJournal of Neurochemistry, 1997
- A bcl-2 transgene expressed in hepatocytes protects mice from fulminant liver destruction but not from rapid death induced by anti-Fas antibody injection.The Journal of Experimental Medicine, 1996
- Bcl-2 Prevents Killing of Neuronal Cells by Glutamate but Not by Amyloid β ProteinBiochemical and Biophysical Research Communications, 1993
- Optimized survival of hippocampal neurons in B27‐supplemented neurobasal™, a new serum‐free medium combinationJournal of Neuroscience Research, 1993
- Excitotoxic cell deathJournal of Neurobiology, 1992
- Neurotrophic and Neurotoxic Effects of Amyloid β Protein: Reversal by Tachykinin NeuropeptidesScience, 1990
- Glutamate toxicity in a neuronal cell line involves inhibition of cystine transport leading to oxidative stressNeuron, 1989
- Neurotoxicity of excitatory amino acid receptor agonists in rat cerebellar slices: Dependence on calcium concentrationNeuroscience Letters, 1986