Trimethyltin-Activated Cyclooxygenase Stimulates Tumor Necrosis Factor-α Release from Glial Cells through Reactive Oxygen Species
- 15 April 2001
- journal article
- Published by Elsevier in Toxicology and Applied Pharmacology
- Vol. 172 (2) , 93-97
- https://doi.org/10.1006/taap.2001.9136
Abstract
No abstract availableKeywords
This publication has 24 references indexed in Scilit:
- Sources of Reactive Oxygen Species Production in Excitotoxin-Stimulated Cerebellar Granule CellsBiochemical and Biophysical Research Communications, 1999
- Trimethyltin but not triethyltin induces specific neural cell death through the protein stanninNeuroscience Research Communications, 1998
- Glia Increase Degeneration of Hippocampal Neurons through Release of Tumor Necrosis Factor-αToxicology and Applied Pharmacology, 1998
- Microglia: a sensor for pathological events in the CNSTrends in Neurosciences, 1996
- Inhibition of the arachidonic acid pathway prevents induction of IL‐8 mRNA by phorbol ester and changes the release of IL‐8 from HL 60 cells: Differential inhibition of induced expression of IL‐8, TNF‐α, IL‐1α, and IL‐1βJournal of Cellular Physiology, 1995
- Trimethyltin increases interleukin (IL)-lα, IL-6 and tumor necrosis factor α mRNA levels in rat hippocampusJournal of Neuroimmunology, 1995
- Effects of Organotins on Rat Brain Astrocytes in CultureJournal of Neurochemistry, 1994
- Elevated central nervous system prostaglandins in human immunodeficiency virus—associated dementiaAnnals of Neurology, 1994
- Polycyclic aromatic hydrocarbon oxidation during prostaglandin biosynthesisLife Sciences, 1981
- Accumulation of Cyclooxygenase Products of Arachidonic Acid Metabolism in Gerbil Brain During Reperfusion After Bilateral Common Carotid Artery OcclusionJournal of Neurochemistry, 1980