The effect of acute and chronic treatment with SCH 23390 on the spontaneous activity of midbrain dopamine neurons
- 1 March 1989
- journal article
- research article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 162 (1) , 109-113
- https://doi.org/10.1016/0014-2999(89)90609-2
Abstract
No abstract availableThis publication has 12 references indexed in Scilit:
- Effect of chronic treatment with SCH 23390 and haloperidol on spontaneous activity of dopamine neurones in substantia nigra pars compacta (SNC) and ventral tegmental area (VTA) in ratsEuropean Journal of Pharmacology, 1988
- The actions of SCH 23390, a D1 receptor antagonist, on operant and avoidance behavior in ratsPharmacology Biochemistry and Behavior, 1987
- SCH 23390, a selective D1 dopamine receptor blocker, enhances the firing rate of nigral dopaminergic neurons but fails to activate striatal tyrosine hydroxylaseBrain Research, 1985
- SCH 23390, a selective dopamine D1 antagonist, activates dopamine neurons but fails to prevent their inhibition by apomorphineEuropean Journal of Pharmacology, 1985
- Antipsychotic drug effects on the electrical activity of dopaminergic neuronsTrends in Neurosciences, 1984
- Pharmacological effects of a specific dopamine D-1 antagonist SCH 23390 in comparison with neurolepticsLife Sciences, 1984
- Differential Effects of Classical and Atypical Antipsychotic Drugs on A9 and A10 Dopamine NeuronsScience, 1983
- Typical and atypical neuroleptics: differential effects of chronic administration on the activity of A9 and A10 midbrain dopaminergic neuronsJournal of Neuroscience, 1983
- The Classification of Dopamine Receptors: Relationship to Radioligand BindingAnnual Review of Neuroscience, 1983
- SCH-23390, A POTENTIAL BENZAZEPINE ANTI-PSYCHOTIC WITH UNIQUE INTERACTIONS ON DOPAMINERGIC SYSTEMS1983