CDK Inhibitor p21 Is Degraded by a Proliferating Cell Nuclear Antigen-coupled Cul4-DDB1Cdt2 Pathway during S Phase and after UV Irradiation

This publication has 43 references indexed in Scilit:

• Ubiquitin- and ATP-Independent Proteolytic Turnover of p21 by the REGγ-Proteasome Pathway
  Molecular Cell, 2007
• Glycogen Synthase Kinase 3β Phosphorylates p21^WAF1/CIP1 for Proteasomal Degradation after UV Irradiation
  Molecular and Cellular Biology, 2007
• Proliferating cell nuclear antigen (PCNA): a dancer with many partners
  Journal of Cell Science, 2003
• CUL-4 ubiquitin ligase maintains genome stability by restraining DNA-replication licensing
  Nature, 2003
• A p53-Dependent Checkpoint Pathway Prevents Rereplication
  Molecular Cell, 2003
• Control of DNA replication licensing in a cell cycle
  Genes to Cells, 2002
• The Human Licensing Factor for DNA Replication Cdt1 Accumulates in G1 and Is Destabilized after Initiation of S-phase
  Journal of Biological Chemistry, 2001
• A degradation signal located in the C-terminus of p21WAF1/CIP1 is a binding site for the C8 alpha-subunit of the 20S proteasome
  The EMBO Journal, 2001
• Proteasomal Turnover of p21Cip1 Does Not Require p21Cip1 Ubiquitination
  Molecular Cell, 2000
• SKP2 is required for ubiquitin-mediated degradation of the CDK inhibitor p27
  Nature Cell Biology, 1999