Calcium Influx through Calcium Leak Channels Is Responsible for the Elevated Levels of Calcium-dependent Proteolysis in Dystrophic Myotubes

This publication has 47 references indexed in Scilit:

• No alteration in gene expression of components of the ubiquitin proteasome proteolytic pathway in dystrophin-deficient muscles
  FEBS Letters, 1996
• The Role of Calpastatin (the Specific Calpain Inhibitor) in Myoblast Differentiation and Fusion
  Biochemical and Biophysical Research Communications, 1996
• Regulatory subunit complex dissociated from 26S proteasome: isolation and characterization
  Biochimica et Biophysica Acta (BBA) - General Subjects, 1995
• Cell Membrane Resealing by a Vesicular Mechanism Similar to Neurotransmitter Release
  Science, 1994
• Cell‐penetrating inhibitors of calpain block both membrane fusion and filamin cleavage in chick embryonic myoblasts
  FEBS Letters, 1993
• Increased Activity of Calcium Leak Channels in Myotubes of Duchenne Human and mdx Mouse Origin
  Science, 1990
• Synthesis of a new cell penetrating calpain inhibitor (calpeptin)
  Biochemical and Biophysical Research Communications, 1988
• Strategies in the Design of Solution-Stable, Water-Soluble Prodrugs II: Properties of Micellar Prodrugs of Methylprednisolone
  Journal of Pharmaceutical Sciences, 1985
• Accumulation of autophagosomes after inhibition of hepatocytic protein degradation by vinblastine, leupeptin or a lysosomotropic amine
  Experimental Cell Research, 1982
• The starvation induced increase in muscle protein degradation is non-lysosomal in origin
  Biochemical and Biophysical Research Communications, 1979