Increased Susceptibility to Apoptosis of Human Hepatocarcinoma Cells Transfected with Antisense N-Acetylglucosaminyltransferase V cDNA

This publication has 19 references indexed in Scilit:

• Elevated activity of N -acetylglucosaminyltransferase V in human hepatocellular carcinoma
  Zeitschrift für Krebsforschung und Klinische Onkologie, 1998
• Alteration of N-acetylglucosaminyltransferases in pancreatic carcinoma.
  Glycoconjugate Journal, 1998
• Inhibition of tumorigenicity in lung adenocarcinoma cells by c-erbB-2 antisense expression
  International Journal of Cancer, 1997
• Regulation of N-acetylglucosaminyltransferase V and Asn-linked oligosaccharide β(1,6) branching by a growth factor signaling pathway and effects on cell adhesion and metastatic potential
  Glycoconjugate Journal, 1997
• Apoptosis induced by retinoic acid in Hep 3B cells in vitro
  Cancer Letters, 1996
• Potentiation of Apoptosis by Treatment With the Protein Kinase C-Specific Inhibitor Safingol in Mitomycin C-Treated Gastric Cancer Cells
  JNCI Journal of the National Cancer Institute, 1995
• Reduced contact-inhibition and substratum adhesion in epithelial cells expressing GlcNAc-transferase V.
  The Journal of cell biology, 1995
• Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation.
  The Journal of cell biology, 1992
• Control of glycoprotein synthesis. The use of oligosaccharide substrates and HPLC to study the sequential pathway for N-acetylglucosaminyltransferases I, II, III, IV, V, and VI in the biosynthesis of highly branched N-glycans by hen oviduct membranes
  Biochemistry and Cell Biology, 1988
• β1-6 Branching of Asn-Linked Oligosaccharides Is Directly Associated with Metastasis
  Science, 1987