Cocaine-Induced Increase in the Permeability Function of Human Vascular Endothelial Cell Monolayers
- 1 June 1999
- journal article
- Published by Elsevier in Experimental and Molecular Pathology
- Vol. 66 (2) , 109-122
- https://doi.org/10.1006/exmp.1999.2253
Abstract
No abstract availableKeywords
This publication has 49 references indexed in Scilit:
- cGMP and Nitric Oxide Modulate Thrombin-Induced Endothelial PermeabilityCirculation Research, 1995
- Molecular and cellular properties of PECAM-1 (endoCAM/CD31): a novel vascular cell-cell adhesion molecule.The Journal of cell biology, 1991
- Alleviation of cocaine-induced coronary vasoconstriction by nitroglycerinJournal of the American College of Cardiology, 1991
- Cocaine Addiction: Psychology and NeurophysiologyScience, 1991
- Cocaethylene: A Unique Cocaine Metabolite Displays High Affinity for the Dopamine TransporterJournal of Neurochemistry, 1991
- Cocaine use and its effect on umbilical artery prostacyclin productionProstaglandins, 1990
- Quantitative analysis of amounts of coronary arterial narrowing in cocaine addictsThe American Journal of Cardiology, 1990
- Thrombin‐induced increase in albumin permeability across the endotheliumJournal of Cellular Physiology, 1986
- Transcellular transport of lipoprotein through arterial endothelial cells in monolayer culture.Cell Structure and Function, 1986
- The Urinary Excretion of Cocaine and Metabolites in Humans: A Kinetic Analysis of Published Data*Journal of Analytical Toxicology, 1985