Exit from G1 and S Phase of the Cell Cycle Is Regulated by Repressor Complexes Containing HDAC-Rb-hSWI/SNF and Rb-hSWI/SNF
Top Cited Papers
- 1 March 2000
- Vol. 101 (1) , 79-89
- https://doi.org/10.1016/s0092-8674(00)80625-x
Abstract
No abstract availableKeywords
This publication has 71 references indexed in Scilit:
- Active Transcriptional Repression by the Rb–E2F Complex Mediates G1 Arrest Triggered by p16INK4a, TGFβ, and Contact InhibitionCell, 1999
- ALTERATION OF NUCLEOSOME STRUCTURE AS A MECHANISM OF TRANSCRIPTIONAL REGULATIONAnnual Review of Biochemistry, 1998
- SWI/SNF Stimulates the Formation of Disparate Activator-Nucleosome Complexes but Is Partially Redundant with Cooperative BindingJournal of Biological Chemistry, 1997
- Mechanism of active transcriptional repression by the retinoblastoma proteinNature, 1995
- The retinoblastoma protein and cell cycle controlCell, 1995
- DP and E2F proteins: coordinating transcription with cell cycle progressionCurrent Opinion in Cell Biology, 1994
- Nucleosome disruption and enhancement of activator binding by a human SW1/SNF complexNature, 1994
- Inhibition of cell proliferation by p107, a relative of the retinoblastoma protein.Genes & Development, 1993
- Retinoblastoma protein switches the E2F site from positive to negative elementNature, 1992
- Identification of a growth suppression domain within the retinoblastoma gene product.Genes & Development, 1992