Transforming growth factor-beta activated during exercise in brain depresses spontaneous motor activity of animals. Relevance to central fatigue

This publication has 22 references indexed in Scilit:

• Release of a Substance that Suppresses Spontaneous Motor Activity in the Brain by Physical Exercise
  Physiology & Behavior, 1998
• Possible mechanisms of central nervous system fatigue during exercise
  Medicine & Science in Sports & Exercise, 1997
• Elevation of Bioactive Transforming Growth Factor-β in Serum from Patients with Chronic Fatigue Syndrome
  Journal of Clinical Immunology, 1997
• Potentiation of Tentacle Ball Formation by a Trypsin-Like Protease and Accompanying Augmented Ingestion in Glutathione-Induced Feeding in Hydra
  Zoological Science, 1995
• Neuroendocrine and substrate responses to altered brain 5-HT activity during prolonged exercise to fatigue
  Journal of Applied Physiology, 1993
• Effect of increased brain serotonergic activity on endurance performance in the rat
  Acta Physiologica Scandinavica, 1992
• Altered cytokine release in peripheral blood mononuclear cell cultures from patients with the chronic fatigue syndrome
  Cytokine, 1991
• Effect of sustained exercise on plasma amino acid concentrations and on 5‐hydroxytryptamine metabolism in six different brain regions in the rat
  Acta Physiologica Scandinavica, 1989
• Changes in plasma concentrations of aromatic and branched‐chain amino acids during sustained exercise in man and their possible role in fatigue
  Acta Physiologica Scandinavica, 1988
• Platelet proteins, including platelet-derived growth factor, specifically depress a subset of the multiple components of the response elicited by glutathione in Hydra.
  The Journal of cell biology, 1987