Abrogation of Nerve Growth Factor-induced Terminal Differentiation by ret Oncogene Involves Perturbation of Nuclear Translocation of ERK
Open Access
- 1 June 2000
- journal article
- Published by Elsevier
- Vol. 275 (25) , 19306-19314
- https://doi.org/10.1074/jbc.275.25.19306
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Cell scattering of SK-N-MC neuroepithelioma cells in response to Ret and FGF receptor tyrosine kinase activation is correlated with sustained ERK2 activationOncogene, 1997
- Distinct biological properties of two RET isoforms activated by MEN 2A and MEN 2B mutationsOncogene, 1997
- Glial Cell Line-derived Neurotrophic Factor Signals through the RET Receptor and Activates Mitogen-activated Protein KinaseJournal of Biological Chemistry, 1996
- RET oncogeneCurrent Opinion in Genetics & Development, 1996
- A Novel Cytoplasmic Dual Specificity Protein Tyrosine Phosphatase Implicated in Muscle and Neuronal DifferentiationJournal of Biological Chemistry, 1996
- Inactivation of p42 MAP kinase by protein phosphatase 2A and a protein tyrosine phosphatase, but not CL100, in various cell linesCurrent Biology, 1995
- Catalytic specificity of protein-tyrosine kinases is critical for selective signallingNature, 1995
- Transformation of Mammalian Cells by Constitutively Active MAP Kinase KinaseScience, 1994
- Inherited cancers associated with the RET proto-oncogeneCurrent Opinion in Genetics & Development, 1994
- Mutations in the RET proto-oncogene are associated with MEN 2A and FMTCHuman Molecular Genetics, 1993