Acute Ethanol Inhibits Extracellular Signal-Regulated Kinase, Protein Kinase B, and Adenosine 3′:5′-Cyclic Monophosphate Response Element Binding Protein Activity in an Age- and Brain Region-Specific Manner
- 1 April 2005
- journal article
- website
- Published by Wiley in Alcohol, Clinical and Experimental Research
- Vol. 29 (4) , 672-682
- https://doi.org/10.1097/01.alc.0000158935.53360.5f
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Cocaine-Induced Intracellular Signaling and Gene Expression Are Oppositely Regulated by the Dopamine D1 and D3 ReceptorsJournal of Neuroscience, 2004
- Alcohol Withdrawal Increases Neuropeptide Y Immunoreactivity in Rat BrainAlcohol, Clinical and Experimental Research, 2003
- The Decreased Phosphorylation of Cyclic Adenosine Monophosphate (cAMP) Response Element Binding (CREB) Protein in the Central Amygdala Acts as a Molecular Substrate for Anxiety Related to Ethanol Withdrawal in RatsAlcohol, Clinical and Experimental Research, 2003
- Ethanol exposure enhances cell death in the developing cerebral cortex: Role of brain‐derived neurotrophic factor and its signaling pathwaysJournal of Neuroscience Research, 2002
- N-Methyl d-Aspartate Receptor-mediated Bidirectional Control of Extracellular Signal-regulated Kinase Activity in Cortical Neuronal CulturesJournal of Biological Chemistry, 2001
- Ethanol-Induced Apoptotic Neurodegeneration and Fetal Alcohol SyndromeScience, 2000
- Chronic Ethanol Exposure Impairs Phosphorylation of CREB and CRE‐Binding Activity in Rat StriatumAlcohol, Clinical and Experimental Research, 1998
- Cell Death During Development Of The Nervous SystemAnnual Review of Neuroscience, 1991
- Effects of Ethanol Exposure during the Third Trimester Equivalent on Neuron Number in Rat Hippocampus and Dentate GyrusAlcohol, Clinical and Experimental Research, 1986
- RECOGNITION OF THE FETAL ALCOHOL SYNDROME IN EARLY INFANCYThe Lancet, 1973