Amyotrophic lateral sclerosis: Recent insights from genetics and transgenic mice
- 1 March 1995
- Vol. 80 (5) , 687-692
- https://doi.org/10.1016/0092-8674(95)90346-1
Abstract
No abstract availableThis publication has 30 references indexed in Scilit:
- Motor neurone disease and animal modelsNeurobiology of Disease, 1994
- Variants of the heavy neurofilament subunit are associated with the development of amyotrophic lateral sclerosisHuman Molecular Genetics, 1994
- Analysis of the functional effects of a mutation in SOD1 associated with familial amyotrophic lateral sclerosisNeuron, 1994
- Oxidative Stress, Glutamate, and Neurodegenerative DisordersScience, 1993
- Amyotrophic Lateral Ssclerosis and Structural Defects in Cu,Zn Superoxide DismutaseScience, 1993
- Parvalbumin and calbindin D‐28k in the human motor system and in motor neuron diseaseNeuropathology and Applied Neurobiology, 1993
- Progressive neuronopathy in transgenic mice expressing the human neurofilament heavy gene: A mouse model of amyotrophic lateral sclerosisCell, 1993
- Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosisNature, 1993
- Hippocampal Zinc Thionein and Pyridoxal Phosphate Modulate Synaptic FunctionsaAnnals of the New York Academy of Sciences, 1990
- Indices of free radical activity in the cerebrospinal fluid in motor neuron disease.Journal of Neurology, Neurosurgery & Psychiatry, 1987