FGF19 as a Postprandial, Insulin-Independent Activator of Hepatic Protein and Glycogen Synthesis
Top Cited Papers
- 25 March 2011
- journal article
- other
- Published by American Association for the Advancement of Science (AAAS) in Science
- Vol. 331 (6024) , 1621-1624
- https://doi.org/10.1126/science.1198363
Abstract
Fibroblast growth factor 19 regulates liver metabolism through a mechanism distinct from that of insulin.Keywords
Funding Information
- National Institutes of Health (DK67158, DK62434, DK40936, U24 DK076169)
This publication has 26 references indexed in Scilit:
- Bifurcation of insulin signaling pathway in rat liver: mTORC1 required for stimulation of lipogenesis, but not inhibition of gluconeogenesisProceedings of the National Academy of Sciences, 2010
- Alterations in Postprandial Hepatic Glycogen Metabolism in Type 2 DiabetesDiabetes, 2004
- Mnk2 and Mnk1 Are Essential for Constitutive and Inducible Phosphorylation of Eukaryotic Initiation Factor 4E but Not for Cell Growth or DevelopmentMolecular and Cellular Biology, 2004
- Fibroblast Growth Factor 19 Increases Metabolic Rate and Reverses Dietary and Leptin-Deficient DiabetesEndocrinology, 2004
- Definition of a novel growth factor-dependent signal cascade for the suppression of bile acid biosynthesisGenes & Development, 2003
- The renaissance of GSK3Nature Reviews Molecular Cell Biology, 2001
- eIF4 Initiation Factors: Effectors of mRNA Recruitment to Ribosomes and Regulators of TranslationAnnual Review of Biochemistry, 1999
- Direct assessment of liver glycogen storage by 13C nuclear magnetic resonance spectroscopy and regulation of glucose homeostasis after a mixed meal in normal subjects.Journal of Clinical Investigation, 1996
- Mitogen inactivation of glycogen synthase kinase-3β in intact cells via serine 9 phosphorylationBiochemical Journal, 1994
- Inactivation of glycogen synthase kinase-3β by phosphorylation: new kinase connections in insulin and growth-factor signallingBiochemical Journal, 1993