Activation of PI3-Kinase Is Required for AMPA Receptor Insertion during LTP of mEPSCs in Cultured Hippocampal Neurons
- 1 May 2003
- Vol. 38 (4) , 611-624
- https://doi.org/10.1016/s0896-6273(03)00228-9
Abstract
No abstract availableKeywords
Funding Information
- Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung (3100-050506.97)
- EJLB Foundation
- Heart and Stroke Foundation of British Columbia and Yukon (T-4824)
- Canadian Institutes of Health Research
This publication has 52 references indexed in Scilit:
- Synaptic plasticity: LTP and LTDPublished by Elsevier ,2003
- Subunit-specific temporal and spatial patterns of AMPA receptor exocytosis in hippocampal neuronsNature Neuroscience, 2001
- Spatially and Functionally Distinct Roles of the PI3-K Effector Pathway during NGF Signaling in Sympathetic NeuronsNeuron, 2000
- Driving AMPA Receptors into Synapses by LTP and CaMKII: Requirement for GluR1 and PDZ Domain InteractionScience, 2000
- Long-term potentiation in dentate gyrus of the rat is inhibited by the phosphoinositide 3–kinase inhibitor, wortmanninNeuropharmacology, 2000
- Insulin-mediated Targeting of Phosphatidylinositol 3-Kinase to GLUT4-containing VesiclesPublished by Elsevier ,1996
- Potential sites of PI-3 kinase function in the endocytic pathway revealed by the PI-3 kinase inhibitor, wortmannin.The Journal of cell biology, 1996
- Long-Term Depression: A Learning-Related Type of Synaptic Plasticity in the Mammalian Central Nervous SystemReviews in the Neurosciences, 1995
- Insulin resistance, diabetes, and the insulin-regulated trafficking of GLUT-4.The Journal of cell biology, 1994
- Properties of vertebrate glutamate receptors: Calcium mobilization and desensitizationProgress in Neurobiology, 1992