The Bare Lymphocyte Syndrome: Molecular Clues to the Transcriptional Regulation of Major Histocompatibility Complex Class II Genes
- 1 August 1999
- journal article
- review article
- Published by Elsevier in American Journal of Human Genetics
- Vol. 65 (2) , 279-286
- https://doi.org/10.1086/302519
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health (AI34000, GM47310, HD34440)
This publication has 66 references indexed in Scilit:
- Major histocompatibility complex class II deficiency: a clinical reviewBlood Reviews, 1996
- Mice Lacking the MHC Class II Transactivator (CIITA) Show Tissue-Specific Impairment of MHC Class II ExpressionPublished by Elsevier ,1996
- Molecular analysis of G1B and G3A IFNγ mutants reveals that defects in CIITA or RFX result in defective class II MHC and li gene inductionImmunity, 1994
- Class II transactivator (CIITA) is sufficient for the inducible expression of major histocompatibility complex class II genes.The Journal of Experimental Medicine, 1994
- Requirement for Protein Synthesis in Antigen Processing by B CellsCellular Immunology, 1994
- Regulation of MHC Class II Expression by Interferon-γ Mediated by the Transactivator Gene CIITAScience, 1994
- Major histocompatibility complex class II deficiency: Clinical manifestations, immunologic features, and outcomeThe Journal of Pediatrics, 1993
- Reactivation of a major histocompatibility complex class II gene in mouse plasmacytoma cells and mouse T cells.The Journal of Experimental Medicine, 1992
- Segregation of MHC class II molecules from MHC class I molecules in the Golgi complex for transport to lysosomal compartmentsNature, 1991
- Congenital immunodeficiency with a regulatory defect in MHC class II gene expression lacks a specific HLA-DR promoter binding protein, RF-XCell, 1988