CpG island hypermethylation is maintained in human colorectal cancer cells after RNAi-mediated depletion of DNMT1
Open Access
- 23 May 2004
- journal article
- research article
- Published by Springer Nature in Nature Genetics
- Vol. 36 (6) , 582-584
- https://doi.org/10.1038/ng1365
Abstract
No abstract availableKeywords
This publication has 15 references indexed in Scilit:
- GATA-4 and GATA-5 Transcription Factor Genes and Potential Downstream Antitumor Target Genes Are Epigenetically Silenced in Colorectal and Gastric CancerMolecular and Cellular Biology, 2003
- DNMT1 is required to maintain CpG methylation and aberrant gene silencing in human cancer cellsNature Genetics, 2002
- Worms gang up on bacteriaNature Reviews Genetics, 2002
- RNA interference: the new somatic cell genetics?Cancer Cell, 2002
- A genomic screen for genes upregulated by demethylation and histone deacetylase inhibition in human colorectal cancerNature Genetics, 2002
- A System for Stable Expression of Short Interfering RNAs in Mammalian CellsScience, 2002
- DNMT1 and DNMT3b cooperate to silence genes in human cancer cellsNature, 2002
- Gene Targeting in Human Cells Without Isogenic DNAScience, 1999
- Methylation-specific PCR: a novel PCR assay for methylation status of CpG islands.Proceedings of the National Academy of Sciences, 1996
- Targeted mutation of the DNA methyltransferase gene results in embryonic lethalityPublished by Elsevier ,1992