3T3 variants unable to bind epidermal growth factor cannot complement in co-culture
- 1 January 1984
- journal article
- research article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 118 (1) , 60-64
- https://doi.org/10.1016/0006-291x(84)91067-2
Abstract
No abstract availableThis publication has 17 references indexed in Scilit:
- Complementation of mutations in the LDL pathway of receptor-mediated endocytosis by cocultivation of LDL receptor-defective hamster cell mutantsCell, 1983
- Biochemical properties of the endothelium‐derived growth factor: Comparision to other growth factorsJournal of Cellular Physiology, 1983
- Glucose uptake and ornithine decarboxylase activity in tetradecanoyl phorbol acetate non-proliferative variantsJournal of Cellular Physiology, 1983
- 12-O-tetradecanoylphorbol-13-acetate stimulates release of arachidonic acid, prostaglandin E2 and prostaglandin F2α from TPA nonproliferative variants of 3T3 cellsBiochemical and Biophysical Research Communications, 1983
- Analysis of phorbol ester receptors in phorbol ester unresponsive 3T3 cell variantsBiochemical and Biophysical Research Communications, 1981
- Isolation of Chinese hamster cell mutants defective in the receptor-mediated endocytosis of low density lipoproteinJournal of Molecular Biology, 1981
- Variants of 3T3 cells lacking mitogenic response to the tumor promoter tetradecanoyl‐phorbol‐acetateJournal of Cellular Physiology, 1981
- Sarcoma growth factor (SGF): Specific binding to epidermal growth factor (EGF) membrane receptorsJournal of Cellular Physiology, 1980
- Mitogenic effects of murine serum and fibroblast growth factor on EGF nonproliferative variants of 3T3Journal of Supramolecular Structure, 1979
- Epidermal Growth Factor and a New DerivativePublished by Elsevier ,1972