Differences on the Inhibitory Specificities of H-Ras, K-Ras, and N-Ras (N17) Dominant Negative Mutants Are Related to Their Membrane Microlocalization
Open Access
- 1 February 2003
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 278 (7) , 4572-4581
- https://doi.org/10.1074/jbc.m209807200
Abstract
No abstract availableKeywords
This publication has 47 references indexed in Scilit:
- Regulating c-Ras function: cholesterol depletion affects caveolin association, GTP loading, and signalingCurrent Biology, 2001
- Maintenance of Cdc42 GDP-bound State by Rho-GDI Inhibits MAP Kinase Activation by the Exchange Factor Ras-GRFJournal of Biological Chemistry, 2001
- Targeted Genomic Disruption of H-ras and N-ras, Individually or in Combination, Reveals the Dispensability of Both Loci for Mouse Growth and DevelopmentMolecular and Cellular Biology, 2001
- The Rho Family GTPase Cdc42 Regulates the Activation of Ras/MAP Kinase by the Exchange Factor Ras-GRFPublished by Elsevier ,2000
- Endogenous c-N-Ras Provides a Steady-state Anti-apoptotic SignalJournal of Biological Chemistry, 2000
- Regulatory Proteins of R-Ras, TC21/R-Ras2, and M-Ras/R-Ras3Journal of Biological Chemistry, 2000
- Cell cycle-dependent activation of RasCurrent Biology, 1996
- Localization of Epidermal Growth Factor-stimulated Ras/Raf-1 Interaction to Caveolae MembraneJournal of Biological Chemistry, 1996
- Localization of Platelet-derived Growth Factor-stimulated Phosphorylation Cascade to CaveolaeJournal of Biological Chemistry, 1996
- Ras-dependent activation of MAP kinase pathway mediated by G-protein βγ subunitsNature, 1994