Glucose Induces MafA Expression in Pancreatic Beta Cell Lines via the Hexosamine Biosynthetic Pathway
Open Access
- 19 January 2007
- journal article
- Published by Elsevier
- Vol. 282 (3) , 1577-1584
- https://doi.org/10.1074/jbc.m605064200
Abstract
No abstract availableKeywords
This publication has 74 references indexed in Scilit:
- FoxA2, Nkx2.2, and PDX-1 Regulate Islet β-Cell-Specific mafA Expression through Conserved Sequences Located between Base Pairs −8118 and −7750 Upstream from the Transcription Start SiteMolecular and Cellular Biology, 2006
- A switch from MafB to MafA expression accompanies differentiation to pancreatic β-cellsDevelopmental Biology, 2006
- Regulation of L-type Ca2+ Channel Activity and Insulin Secretion by the Rem2 GTPasePublished by Elsevier ,2005
- O-GlcNAc modification of nucleocytoplasmic proteins and diabetesMedical Molecular Morphology, 2005
- Roles of maf family proteins in lens developmentDevelopmental Dynamics, 2004
- Mouse MafA, homologue of zebrafish somite Maf 1, contributes to the specific transcriptional activity through the insulin promoterBiochemical and Biophysical Research Communications, 2003
- Insulin feedback action on pancreatic β‐cell functionFEBS Letters, 2002
- Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT MethodMethods, 2001
- Chronic exposure of betaTC-6 cells to supraphysiologic concentrations of glucose decreases binding of the RIPE3b1 insulin gene transcription activator.Journal of Clinical Investigation, 1996
- The reduction of insulin gene transcription in HIT-T15 beta cells chronically exposed to high glucose concentration is associated with the loss of RIPE3b1 and STF-1 transcription factor expressionMolecular Endocrinology, 1995