Anticonvulsants attenuate amyloid β-peptide neurotoxicity, Ca2+ deregulation, and cytoskeletal pathology
- 1 March 1995
- journal article
- research article
- Published by Elsevier in Neurobiology of Aging
- Vol. 16 (2) , 187-198
- https://doi.org/10.1016/0197-4580(94)00150-2
Abstract
No abstract availableKeywords
This publication has 74 references indexed in Scilit:
- β-Amyloid Peptide Free Radical Fragments Initiate Synaptosomal Lipoperoxidation in a Sequence-Specific Fashion: Implications to Alzheimer′s DiseaseBiochemical and Biophysical Research Communications, 1994
- Abnormal Alzheimer‐like phosphorylation of tau‐protein by cyclin‐dependent kinases cdk2 and cdk5Published by Wiley ,1993
- Abnormal tau phosphorylation at Ser396 in alzheimer's disease recapitulates development and contributes to reduced microtubule bindingNeuron, 1993
- Phenytoin prevents stress‐ and corticosterone‐induced atrophy of CA3 pyramidal neuronsHippocampus, 1992
- Glucose deprivation elicits neurofibrillary tangle-like antigenic changes in hippocampal neurons: Prevention by NGF and bFGFExperimental Neurology, 1992
- Vitamin E protects nerve cells from amyloid βprotein toxicityBiochemical and Biophysical Research Communications, 1992
- Tau antigenic changes induced by glutamate in rat primary culture model: A biochemical approachNeuroscience Letters, 1992
- Agonist‐evoked Ca2+ transients in primary astroglial cultures–modulatory effects of valproic acidGlia, 1992
- B- Amyloid increases neuronal susceptibility to injufy by glucose deprivationNeuroReport, 1991
- Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's diseaseNature, 1991