Disruption of Redox Homeostasis in the Transforming Growth Factor-α/c-myc Transgenic Mouse Model of Accelerated Hepatocarcinogenesis
Open Access
- 1 June 1998
- journal article
- research article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 273 (25) , 15846-15853
- https://doi.org/10.1074/jbc.273.25.15846
Abstract
No abstract availableKeywords
This publication has 64 references indexed in Scilit:
- Increased Oxidative Damage to Mitochondrial DNA Following Chronic Ethanol ConsumptionBiochemical and Biophysical Research Communications, 1997
- Quantification of Wild-Type Mitochondrial DNA and Its 4.8-kb Deletion in Rat OrgansBiochemical and Biophysical Research Communications, 1997
- Insulin-induced Activation of NADPH-dependent H2O2 Generation in Human Adipocyte Plasma Membranes Is Mediated by Gαi2Journal of Biological Chemistry, 1997
- c-myc-dependent hepatoma cell apoptosis results from oxidative stress and not a deficiency of growth factorsJournal of Cellular Physiology, 1997
- Age-Associated Alterations of the Mitochondrial GenomeFree Radical Biology & Medicine, 1997
- Genetic analysis of ageing: role of oxidative damage and environmental stressesNature Genetics, 1996
- Reduced Growth Capacity of Hepatocytes from c-myc and c-myc/TGF-α Transgenic Mice in Primary CultureBiochemical and Biophysical Research Communications, 1996
- Role of oxygen free radicals in cancer developmentEuropean Journal Of Cancer, 1996
- Hepatic mitochondrial DNA deletion in alcoholics: Association with microvesicular steatosisGastroenterology, 1995
- Lipid peroxidation and cancerCritical Reviews in Oncology/Hematology, 1993