Mutations in the C, D, and V Open Reading Frames of Human Parainfluenza Virus Type 3 Attenuate Replication in Rodents and Primates
- 1 September 1999
- Vol. 261 (2) , 319-330
- https://doi.org/10.1006/viro.1999.9878
Abstract
No abstract availableKeywords
This publication has 51 references indexed in Scilit:
- The immunogenicity and efficacy of intranasally or parenterally administered replication-deficient vaccinia-parainfluenza virus type 3 recombinants in rhesus monkeysVaccine, 1998
- Sendai Viruses with Altered P, V, and W Protein ExpressionVirology, 1998
- Recovery of Infectious Human Parainfluenza Virus Type 3 from cDNAVirology, 1997
- Minimum Protein Requirements for Transcription and RNA Replication of a Minigenome of Human Parainfluenza Virus Type 3 and Evaluation of the Rule of SixVirology, 1997
- Normal Cellular Replication of Sendai Virus without thetrans-Frame, Nonstructural V ProteinVirology, 1997
- Production of infectious human respiratory syncytial virus from cloned cDNA confirms an essential role for the transcription elongation factor from the 5' proximal open reading frame of the M2 mRNA in gene expression and provides a capability for vaccine development.Proceedings of the National Academy of Sciences, 1995
- The Sendai virus nonstructural C proteins specifically inhibit viral mRNA synthesisVirology, 1992
- RNA editing in the phosphoprotein gene of the human parainfluenza virus type 3Virology, 1992
- Sendai Virus P Gene Produces Multpile Proteins from Overlapping Open Reading FramesEnzyme, 1990
- Measles virus editing provides an additional cysteine-rich proteinCell, 1989