Oxidative stress and kidney dysfunction due to ischemia/reperfusion in rat: Attenuation by dehydroepiandrosterone
- 1 September 2003
- journal article
- Published by Elsevier in Kidney International
- Vol. 64 (3) , 836-843
- https://doi.org/10.1046/j.1523-1755.2003.00152.x
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Dehydroepiandrosterone Activates Endothelial Cell Nitric-oxide Synthase by a Specific Plasma Membrane Receptor Coupled to Gαi2,3Journal of Biological Chemistry, 2002
- Postischemic renal oxidative stress induces an inflammatory response through PAF and oxidized phospholipids: prevention by antioxidant treatmentThe FASEB Journal, 2002
- Comparison of tumor necrosis factor-α effect on the expression of iNOS in macrophage and cardiac myocytesPerfusion, 2001
- Molecular Mechanisms of Tumor Necrosis Factor α Gene Expression in Monocytic Cells via Hyperglycemia-induced Oxidant Stress-dependent and -independent PathwaysJournal of Biological Chemistry, 2000
- Lipopolysaccharide Pretreatment Protects from Renal Ischemia/Reperfusion InjuryThe American Journal of Pathology, 2000
- Hemolytic effects of dehydroepiandrosterone in vitroLife Sciences, 1997
- 5-HPETE Is a Potent Inhibitor of Neuronal Na+, K+-ATPase ActivityBiochemical and Biophysical Research Communications, 1997
- Identification of an NADH plus iron dependent, Ca2+ activated hydrogen peroxide production in synaptosomesBiochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1993
- Control of regional blood flow by endothelium-derived nitric oxide.Hypertension, 1990
- Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4Nature, 1970