Sulindac activates nuclear translocation of AIF, DFF40 and endonuclease G but not induces oligonucleosomal DNA fragmentation in HT-29 cells

This publication has 30 references indexed in Scilit:

• Synthetic chenodeoxycholic acid derivative HS-1200-induced apoptosis of p815 mastocytoma cells is augmented by co-treatment with lactacystin
  Anti-Cancer Drugs, 2003
• The role of mitochondrial factors in apoptosis: a Russian roulette with more than one bullet
  Cell Death & Differentiation, 2002
• Mitochondria and Apoptosis
  Science, 1998
• 7-Hydroxystaurosporine (UCN-01) Induces Apoptosis in Human Colon Carcinoma and Leukemia Cells Independently of p53
  Experimental Cell Research, 1997
• Appearance of 1∼2 Mbp Giant DNA Fragments as an Early Common Response Leading to Cell Death Induced by Various Substances That Cause Oxidative Stress
  Free Radical Biology & Medicine, 1997
• Nonsteroidal Antiinflammatory Drugs Inhibit the Proliferation of Colon Adenocarcinoma Cells: Effects on Cell Cycle and Apoptosis
  Experimental Cell Research, 1996
• Evolutionary conservation of a genetic pathway of programmed cell death
  Journal of Cellular Biochemistry, 1996
• The effects of sulindac on colorectal proliferation and apoptosis in familial adenomatous polyposis
  Gastroenterology, 1995
• Sulindac sulfide, an aspirin-like compound, inhibits proliferation, causes cell cycle quiescence, and induces apoptosis in HT-29 colon adenocarcinoma cells.
  Journal of Clinical Investigation, 1995
• Programmed cell death: Apoptosis and oncogenesis
  Cell, 1991