Neurodegenerative and morphogenic changes in a mouse model of temporal lobe epilepsy do not depend on the expression of the calcium-binding proteins parvalbumin, calbindin, or calretinin
- 13 April 2000
- journal article
- Published by Elsevier in Neuroscience
- Vol. 97 (1) , 47-58
- https://doi.org/10.1016/s0306-4522(00)00017-8
Abstract
No abstract availableKeywords
This publication has 61 references indexed in Scilit:
- Recurrent seizures and hippocampal sclerosis following intrahippocampal kainate injection in adult mice: electroencephalography, histopathology and synaptic reorganization similar to mesial temporal lobe epilepsyNeuroscience, 1999
- Knock-Out Mice Reveal a Critical Antiepileptic Role for Neuropeptide YJournal of Neuroscience, 1997
- Neurotrophin release by neurotrophins: Implications for activity-dependent neuronal plasticityProceedings of the National Academy of Sciences, 1997
- Ataxia and altered dendritic calcium signaling in mice carrying a targeted null mutation of the calbindin D28k geneProceedings of the National Academy of Sciences, 1997
- Distribution, ultrastructure, and connectivity of calretinin-immunoreactive mossy cells of the mouse dentate gyrusHippocampus, 1997
- GABA TRANSPORTER HETEROGENEITY: PHARMACOLOGY AND CELLULAR LOCALIZATIONNeurochemistry International, 1996
- Regulation of neuropeptide expression in the brain by neurotrophinsMolecular Neurobiology, 1995
- Investigations into neuropeptide Y‐mediated presynaptic inhibition in cultured hippocampal neurones of the ratBritish Journal of Pharmacology, 1992
- Parvalbumin in Most γ-Aminobutyric Acid-Containing Neurons of the Rat Cerebral CortexScience, 1986
- Immunohistochemical localization of calcium-binding protein in the cerebellum, hippocampal formation and olfactory bulb of the ratBrain Research, 1982