Deficits in the control of food intake after hypothalamic paraventricular nucleus lesions
- 1 December 1985
- journal article
- research article
- Published by Elsevier in Physiology & Behavior
- Vol. 35 (6) , 883-890
- https://doi.org/10.1016/0031-9384(85)90255-0
Abstract
No abstract availableKeywords
This publication has 14 references indexed in Scilit:
- Macronutrient self-selection in three forms of hypothalamic obesityAmerican Journal of Physiology-Regulatory, Integrative and Comparative Physiology, 1983
- Increased carbohydrate consumption by rats as a function of 2-deoxy-D-glucose administrationPharmacology Biochemistry and Behavior, 1983
- Effects of short-term nocturnal and diurnal food deprivation on subsequent feeding in intact and VMH lesioned rats: Relation to blood glucose levelPhysiology & Behavior, 1982
- Adrenocorticotropic Regulations after Bilateral Lesions of the Paraventricular or Supraoptic Nuclei and in Brattleboro RatsNeuroendocrinology, 1982
- Hypothalamic paraventricular nucleus lesions produce overeating and obesity in the ratPhysiology & Behavior, 1981
- Glucoreceptors Controlling Feeding and Blood Glucose: Location in the HindbrainScience, 1981
- Analysis of behavioral deficits produced by lesions in the dorsal and ventral midbrain tegmentumPhysiology & Behavior, 1980
- Increased carbohydrate intake as a function of insulin administration in rats☆Physiology & Behavior, 1980
- Paraventricular nucleus: A primary site mediating adrenergic stimulation of feeding and drinkingPharmacology Biochemistry and Behavior, 1978
- Specific loss of the hypoglycemic control of feeding in recovered lateral ratsAmerican Journal of Physiology-Legacy Content, 1967