Abstract
One of the most important, if not the principal, factor in peritonitis is the accompanying toxemia. Since the success or failure of treatment depends largely on measures designed either to neutralize the toxins as formed or to prevent their absorption into the systemic circulation, it is natural that a great deal of interest has been evinced in the manner in which these toxins enter the systemic circulation. A survey of the literature reveals that the absorption from the peritoneal cavity is as yet not thoroughly understood. There are many factors which affect the rate of absorption, and there is more than one avenue of escape of material from the peritoneal cavity. While it is not the purpose of this report to comment on how material, whether it is particulate, crystalloid or a colloid solution, leaves the peritoneal cavity, a brief summary of the evidence at hand may be of interest.

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