Innate Immunity: The Bridge between Adaptive Immunity and Inflammation, December 17, 1998, New York Academy of Sciences, New York, NY, USA
- 1 May 1999
- journal article
- abstracts
- Published by Springer Nature in Inflammation Research
- Vol. 48 (5) , 232-235
- https://doi.org/10.1007/s000110050451
Abstract
No abstract availableThis publication has 17 references indexed in Scilit:
- Immune Suppression by Recombinant Interleukin (rIL)-12 Involves Interferon γ Induction of Nitric Oxide Synthase 2 (iNOS) Activity: Inhibitors of NO Generation Reveal the Extent of rIL-12 Vaccine Adjuvant EffectThe Journal of Experimental Medicine, 1998
- Interleukin-12 and interleukin-18 synergistically induce murine tumor regression which involves inhibition of angiogenesis.Journal of Clinical Investigation, 1998
- Mechanisms of innate resistance toToxoplasma gondiiinfectionPhilosophical Transactions Of The Royal Society B-Biological Sciences, 1997
- How Are NK Cell Responses Regulated during Infection?Experimental Parasitology, 1996
- TNF-α AND IL-6 RESPONSE IN GERM-FREE AND GERM BEARING RATS AFTER ENDOTOXIN AND HEMORRHAGIC SHOCK.Shock, 1996
- Role of Tyrosine Kinases, Protein Kinase C, and Protein Kinase A in the Regulation of Interferon-α Production Induced by Herpes Simplex Virus Type 1Journal of Interferon & Cytokine Research, 1996
- A COMPARISON OF SURVIVAL AT DIFFERENT DEGREES OF HEMORRHAGIC SHOCK IN GERM-FREE AND GERM-BEARING RATSShock, 1995
- PULMONARY CYTOKINE GENE EXPRESSION AND ENDOTOXIN LEVELS DIFFER IN GERM-BEARING AND GERM-FREE RATS DURING HEMORRHAGICShock, 1995
- Viral Induction of Low Frequency Interferon-? Producing CellsVirology, 1994
- 53; A RAT INTESTINAL CRYPTDIN-LIKE mRNA IS INCREASED AFTER HEMORRHAGIC SHOCKShock, 1994