Escape from the antiproliferative effect of transforming growth factor-β1 in LLC-PK1 renal epithelial cells
- 1 March 1994
- journal article
- Published by Elsevier in Kidney International
- Vol. 45 (3) , 642-649
- https://doi.org/10.1038/ki.1994.86
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- Negative Regulation of G1 in Mammalian Cells: Inhibition of Cyclin E-Dependent Kinase by TGF-βScience, 1993
- Role of proteoglycans and cytoskeleton in the effects of TGF-β1 on renal proximal tubule cellsKidney International, 1993
- Abrogation by c-myc of Gl phase arrest induced by RB protein but not by p53Nature, 1992
- Amino-terminal domains of c-myc and N-myc proteins mediate binding to the retinoblastoma gene productNature, 1991
- Evidence for c‐myc in the signaling pathway for TGF‐β in well‐differentiated human colon carcinoma cellsJournal of Cellular Physiology, 1990
- TGF-β stimulation and inhibition of cell proliferation: New mechanistic insightsCell, 1990
- Growth inhibition by TGF-β linked to suppression of retinoblastoma protein phosphorylationCell, 1990
- TGF-β1 inhibition of c-myc transcription and growth in keratinocytes is abrogated by viral transforming proteins with pRB binding domainsCell, 1990
- TGF-β inhibition of endothelial cell proliferation: Alteration of EGF binding and EGF-induced growth-regulatory (competence) gene expressionCell, 1987
- Suppression of the EGF-dependent induction of c-myc proto-oncogene expression by transforming growth factor β in a human breast carcinoma cell lineBiochemical and Biophysical Research Communications, 1987