Expanded Polyglutamine Protein Forms Nuclear Inclusions and Causes Neural Degeneration in Drosophila

This publication has 38 references indexed in Scilit:

• Rethinking Genotype and Phenotype Correlations in Polyglutamine Expansion Disorders
  Human Molecular Genetics, 1997
• Aggregation of Huntingtin in Neuronal Intranuclear Inclusions and Dystrophic Neurites in Brain
  Science, 1997
• Formation of Neuronal Intranuclear Inclusions Underlies the Neurological Dysfunction in Mice Transgenic for the HD Mutation
  Cell, 1997
• Reiterative Use of the EGF Receptor Triggers Differentiation of All Cell Types in the Drosophila Eye
  Cell, 1996
• grim, a novel cell death gene in Drosophila.
  Genes & Development, 1996
• Spinocerebellar ataxia 3 and machado‐joseph disease: Clinical, molecular, and neuropathological features
  Annals of Neurology, 1996
• The Haw River Syndrome: Dentatorubropallidoluysian atrophy (DRPLA) in an African–American family
  Nature Genetics, 1994
• The eyes absent gene: Genetic control of cell survival and differentiation in the developing Drosophila eye
  Cell, 1993
• An extensive 3′ cis-regulatory region directs the imaginal disk expression of decapentaplegic, a member of the TGF-β family in Drosophila
  Development, 1991
• The emergence of order in the Drosophila pupal retina
  Developmental Biology, 1989