P2X7 receptor activation causes phosphatidylserine exposure in human erythrocytes
- 30 March 2007
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 355 (1) , 169-173
- https://doi.org/10.1016/j.bbrc.2007.01.124
Abstract
No abstract availableKeywords
This publication has 14 references indexed in Scilit:
- A Thr357 to Ser Polymorphism in Homozygous and Compound Heterozygous Subjects Causes Absent or Reduced P2X7 Function and Impairs ATP-induced Mycobacterial Killing by MacrophagesPublished by Elsevier ,2006
- A 5′ intronic splice site polymorphism leads to a null allele of the P2X7 gene in 1–2% of the Caucasian populationFEBS Letters, 2005
- Extracellular ATP Increases Cation Fluxes in Human Erythrocytes by Activation of the P2X7 ReceptorJournal of Biological Chemistry, 2004
- Involvement of Sodium in Early Phosphatidylserine Exposure and Phospholipid Scrambling Induced by P2X7 Purinoceptor Activation in ThymocytesJournal of Biological Chemistry, 2004
- The Pro-451 to Leu Polymorphism within the C-terminal Tail of P2X7 Receptor Impairs Cell Death but Not Phospholipase D Activation in Murine ThymocytesPublished by Elsevier ,2004
- Role of Ca2+-activated K+ channels in human erythrocyte apoptosisAmerican Journal of Physiology-Cell Physiology, 2003
- Cation channels trigger apoptotic death of erythrocytesCell Death & Differentiation, 2003
- Molecular Physiology of P2X ReceptorsPhysiological Reviews, 2002
- Programmed cell death in mature erythrocytes: a model for investigating death effector pathways operating in the absence of mitochondriaCell Death & Differentiation, 2001
- Preparation of solutions with free calcium concentration in the nanomolar range using 1,2-bis(o-aminophenoxy)ethane-N,N,N′,N′-tetraacetic acidAnalytical Biochemistry, 1991